β-lapachone reduces cell viability, migration, and epithelial–mesenchymal transition in mammary tumor spheroids - Summary - DentalSpire

β-lapachone reduces cell viability, migration, and epithelial–mesenchymal transition in mammary tumor spheroids

  • By

  • Laura Lacerda Coelho

  • Matheus Menezes Vianna

  • Debora Moraes da Silva

  • Beatriz Matheus de Souza Gonzaga

  • Roberto Rodrigues Ferreira

  • Claudia Mara Lara Melo Coutinho

  • Fatima Cristina Mendes Magalhães

  • Edmilson José Maria

  • Rodrigo Rodrigues de Oliveira

  • Pedro Paulo de Abreu Manso

  • Marcelo Alex de Carvalho

  • Fernando Regla Vargas

  • Luciana Ribeiro Garzoni

  • November 26, 2025

  • 0 min

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Objective:

To evaluate the cytotoxic and antimetastatic effects of β-lapachone on mammary tumor spheroids (MTS) and its potential implications for breast cancer treatment.

Key Findings:
  • β-lapachone induced significant cell death in MCF-7 spheroids, suggesting its potential as a therapeutic agent.
  • Cell viability decreased with increasing concentrations of β-lapachone, indicating a dose-dependent effect.
  • Migration of cells from spheroids was suppressed by β-lapachone treatment, highlighting its antimetastatic properties.
  • β-lapachone restored E-cadherin expression and downregulated vimentin, indicating modulation of epithelial–mesenchymal transition (EMT) and its implications for cancer progression.
Interpretation:

The findings suggest that β-lapachone has potential as an antitumor agent by reducing cell viability and migration while affecting EMT markers in breast cancer models, warranting further investigation into its clinical applications.

Limitations:
  • The study was conducted in vitro, which may not fully replicate in vivo conditions; future studies should include in vivo models.
  • Only one breast cancer cell line (MCF-7) was used, limiting the generalizability of the results; additional cell lines should be tested.
Conclusion:

β-lapachone shows promise as a therapeutic agent against breast cancer by inhibiting cell viability and migration, warranting further investigation in clinical settings to explore its full potential.

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